Circulation




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Intermediate Effect Variants in HCM: Integration into Clinical Practice and Family Screening.

doi : 10.1161/CIRCULATIONAHA.125.077435

Volume 153(18) pgs. 1347-1441,e1279-e1297 May 05, 2026

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MR-proADM Predicts Mortality and Heart Failure Events in ATTR Cardiac Amyloidosis.

doi : 10.1161/CIRCULATIONAHA.125.077833

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Multiorgan Physiological Deficits During Exercise Identify Clinical and Molecular Predisposition to Heart Failure With Preserved Ejection Fraction.

doi : 10.1161/CIRCULATIONAHA.125.077579

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P16+ Cells Drive Adverse Postischemic Cardiac Remodeling Through CCL8-Mediated Recruitment of Cytotoxic Lymphocytes.

doi : 10.1161/CIRCULATIONAHA.125.077172

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Small Extracellular Vesicle External Surface Adiponectin-Mediated Adipocytes/Cardiomyocytes Communication in Diabetic Ischemic Heart Failure.

doi : 10.1161/CIRCULATIONAHA.125.076372

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A New Right Bundle Branch Block-Like Rhythm Immediately After TAVR.

doi : 10.1161/CIRCULATIONAHA.126.080374

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Daily Stellate Ganglion Phototherapy for Ventricular Arrhythmias: A Noninvasive Neuromodulation Approach for Outpatient Care.

doi : 10.1161/CIRCULATIONAHA.125.078175

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Letter by Lu et al Regarding Article, "Ivabradine in Patients Undergoing Noncardiac Surgery: A Randomized Controlled Trial".

doi : 10.1161/CIRCULATIONAHA.125.077593

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Letter by Yang and Wu Regarding Article, "Ivabradine in Patients Undergoing Noncardiac Surgery: A Randomized Controlled Trial".

doi : 10.1161/CIRCULATIONAHA.125.077683

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Response by Szczeklik et al to Letter Regarding Article, "Ivabradine in Patients Undergoing Noncardiac Surgery: A Randomized Controlled Trial".

doi : 10.1161/CIRCULATIONAHA.126.079809

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2026 Dietary Guidance to Improve Cardiovascular Health: A Scientific Statement From the American Heart Association.

doi : 10.1161/CIR.0000000000001435

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Retraction of: Abstract 4349903: Exercise can alleviate hypertrophic cardiomyopathy induced by the novel MYH7 T265I mutation.

doi : 10.1161/CIR.0000000000001443

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Retraction of: Abstract 4350116: [beta]1-adrenergic autoantibodies ([beta]1-AA) augment macropinocytosis in CD4+ T cells, leading to the expansion of CD4+CD28- T cell subsets in heart failure.

doi : 10.1161/CIR.0000000000001444

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